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the expression of H3K4me3 was significantly increased, and emphysema inflammation, apoptosis, and oxidative stress levels were more severe in CSE-induced mice

Given that IL-1RA competes with IL-1 for binding to the IL-1 receptor and prevents IL-1-induced pro-inflammatory signalling we wondered if the increased production of IL-1RA in smokers was associated with high IL-1 levels

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That's changed over the last 50 years as tobacco advertising disappeared from the airwaves and a massive health campaign discouraged Americans from smoking