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SL inhibited colocalization of NLRP3 and apoptosis-associated speck-like protein containing a caspase-recruitment domain (ASC) as well as -tubulin and acetylated--tubulin in response to palmitate treatment, explaining that the NAD+/SIRT2 pathway was an important mediator through which SL prevented NLRP3 inflammasome activation during NAFLD (Zhang et al., 2018)

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These medications can influence digestion and appetite and may contribute to changes in hydration and lean muscle mass during weight loss

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