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ROS can further activate epithelial cells and macrophages facilitating neutrophil, monocyte, and lymphocyte recruitment, and the recruited activated inflammatory cells subsequently enhance additional ROS generation, increasing the pro-oxidant burden (8085)

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Notably, ACSS1 can catalyze the production of acetyl-CoA from acetic acid in immune cells within the tumor microenvironment, thereby promoting the proliferation of T cells and NK cells and contributing to the clearance of tumor cells [48]