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However, when KA is oriented at the entrance of the active site, it restricts substrate entrance and product efflux, consequently, TyrBm cannot reach its maximum velocity (Table 1)

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3.7 Effect of glutathione administration on endothelium-dependent relaxation of isolated vascular rings Figure 5A shows typical records of relaxation curves of vascular preparations due to different doses of acetylcholine application against the background of norepinephrine-induced vasoconstriction of glutathione-treated and untreated old rats