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Atypical COL3A1 variants (glutamic acid to lysine) cause vascular EhlersDanlos syndrome with a consistent phenotype of tissue fragility and skin hyperextensibility

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[2] [3] NAD+ is continuously recycled within cells as it is interconverted to and from its various forms through salvage pathways

Fluorescence was measured 60 min after treatment with 2-deoxyglucose 6-phosphate using GloMax discover (Promega)