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It also regulates the protein expression of TGF-1 (a key pro-fibrotic factor), TNF-, and decorin (an anti-fibrotic agent), inhibits HSCs activation and reduces extracellular matrix (ECM) accumulation, ultimately demonstrating its hepatoprotective activity (Wang et al., 2018)

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Crucially, FOXO4 knockdown significantly reduced the viability of senescent TM3 Leydig cells and substantially increased their rate of apoptosis