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Unlike whole-cell therapies, MSC-Exos mitigate risks associated with tumor formation and immune rejection while maintaining the regenerative and anti-inflammatory properties of their parent cells [10]

In addition, BPC 157 may counteract the disturbances of gap junction [107] postulated in cell death and neuromodulation in the retina [137] as BPC 157 acts as a stabilizer of the cellular junction [107]

Mechanism of Action: IGF-1 LR3 vs IGF-1 DES Both IGF-1 LR3 and IGF-1 DES bind to the IGF-1 receptor , triggering powerful anabolic pathwaysprimarily PI3K/Akt/mTOR for muscle protein synthesis and MAPK for cell proliferation