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Indeed, chronic alcoholism and nicotine consumption are independent risk factors for pancreatitis, and both inhibit pancreatic thiamine uptake via downregulation of thiamine transporters (90)

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The protective effects of probiotics are largely attributed to their capacity to suppress urease-producing, pro-inflammatory pathogenic bacteria, thereby reducing the risk of HE development (499)

The use of nicotinamide riboside plus l-carnitine in high-fat-diet-treated mice with non-alcoholic fatty liver disease induced anti-obesogenic hepatoprotective activity due to regulation of INSR, PPARGC1B, SREBF, SCAP, and ACOX, as well as reduction of hepatic steatosis, fat mass, and obesity [77]