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The priming signal involves the upregulation of NLRP3 expression and the synthesis of proinflammatory cytokines such as TNF- and IL-1, typically mediated by nuclear factor kappa B activation in response to inflammatory stimuli
Stacking two GLP-1 class compounds (e.g., semaglutide + tirzepatide) is not supported by any clinical trial, is likely to produce additive GI side effects, and has no mechanistic rationale the receptors overlap too much
Ishigaki et al., 2007), thus suggesting a role also in non-neuronal cells
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