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In the parasite, TGR completely replaces more specialized TrxR and GR orthologs and, therefore, TGR functions in reducing both Trx and GSSG

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Dopamine-induced oxidative stress, impaired synaptic vesicle function and misfolding of -synuclein, due to mutations or to oxidative damage to this protein, might be components in a self-perpetuating vicious cycle that eventually leads to the demise of dopaminergic neurons