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Studies of ascorbate/DHA breakdown products have to a large extent been carried out under apoplastic conditions and with exogenously-supplied 14 C-ascorbate

And that comparison uses the enhanced version of phentermine, not phentermine alone, which performs worse

Key limitations of current evidence include: Relatively short follow-up periods (typically 35 years) in randomised trials, whereas pancreatic cancer may take years to develop Potential for detection bias, as patients on GLP-1 therapy may undergo more frequent medical monitoring Difficulty accounting for all confounding factors in observational studies Despite these limitations, the weight of current evidence does not support a causal link between GLP-1 receptor agonist use and pancreatic cancer development

All that WADA has to do is (a) reclassify the Section 4 Substances as Threshold Substances (or otherwise treat them the same), and (b) allow the concentration of the relevant substance in the B sample to be calculated and taken into account to determine whether an anti-doping rule violation should be pursued