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[DOI] [PubMed] [Google Scholar] 54.Bahmad H.F., Jalloul M., Azar J., Moubarak M.M., Samad T.A., Mukherji D., Al-Sayegh M., Abou-Kheir W

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It enhances the efficacy of existing AML therapies and provides a promising combination treatment approach that exploits mitochondrial vulnerability and metabolic reprogramming to improve treatment outcomes in AML

The human genome encodes up to 17 genes of cytosolic (soluble) GSTs, but two of them are frequently represented by null alleles, GSTM1*0 and GSTT1*0 , such that the affected individuals do not express the corresponding enzymes 9