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The neurotoxic effects of mHtt are mediated by the induction of mitochondrial defects, which in turn result in aberrant energy metabolism and HD-related neuronal dysfunction ( Studies have demonstrated that mHTT interacts with the PGC-1 promoter, directly impairs the activity of PGC-1, and impedes the capacity of PGC-1 to open downstream target genes (Sharma A

Mitapivat markedly reduced erythrophagocytosis in spleens of 4.2 / mice when compared with 4.2 / controls (Figure 4B)

In general, mycotoxins effects on DNA, RNA, protein synthesis and the pro-apoptotic action causing changes in physiological functions including growth, development and reproduction [2]

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