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The finding that only DFO prevented LCN2-induced death supports a mechanism of iron-dependent oxidative damage consistent with ferroptosis

Both PDGF-BB and BPC-157 were observed to exhibit similar selectivity for stimulating granulation tissue in both models

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Therefore, GLP-1R stimulation may activate the -catenin pathway, which may result in a concerted action by TCF4 and FOXA1 to regulate the expression of FABP1 and hence prevent the lipid accumulation induced by OA (Fig