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This interaction initiates the transcription of a broad array of cytoprotective genes, markedly enhancing the synthesis of intracellular glutathione and upregulating phase II detoxification and antioxidant enzymes, including glutathione S-transferases (GSTs) and heme oxygenase-1 (HO-1)

Mouse studies have shown that mTOR inhibitors such as Rapamycin, which formerly received FDA approval for cancer treatments, successfully rescued deficiencies in social and repetitive behaviors in postnatal day 7 TSC-mutant mice, and only social deficits in 6-week-old TSC-mutant mice [118, 119]