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Similar to Rac1, the increased Cdc42 activity was colocalized with actin filaments and the inhibition of Cdc42 resulted in decreased actin filaments, supporting the involvement of actin changes in AD pathogenesis (Mendoza-Naranjo et al., 2007)
Blood was drawn from the inferior vena cava and allowed to clot, and then serum was collected by centrifugation at 14,000 g for 10 min
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These limitations may partially explain the discrepancy between promising preclinical results and the limited success observed in clinical trials
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