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We are studying resistance to estrogen induced apoptosis because clinical experience shows us that only 30% of patients respond to estrogen induced apoptosis once exhaustive antihormonal therapy occurs [9]

NAC treatment, however, caused only moderate recovery of aspirin-induced apoptosis in HepG2 cells suggesting additional factor(s), other than altered GSH pool, which could play a role in inducing apoptosis by ASA

Peripheral blood mononuclear cells (PBMC) were isolated by leukapheresis and stored in liquid nitrogen