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The neuroprotective mechanisms may involve modulation of inflammatory responses within the central nervous system, promotion of neuronal survival, and enhancement of axonal regeneration and synaptic plasticity

The concept works like this: by presenting the body's own peptide epitopes, the fragments of self-proteins that T cells recognize, in a tolerogenic context, researchers can selectively delete or suppress the specific T cell clones responsible for autoimmune attack

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Such complaints have been described in literature from 1900 onwards and may precede abnormal blood counts