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Additionally, Dex has demonstrated its ability to inhibit apoptosis induced by oxidative stress while promoting the repair of synaptic damage by inhibiting the interaction between N-Methyl-D-aspartic acid (NMDA) and postsynaptic density protein 95 (PSD95), thus reducing the formation of NMDA-PSD95 complexes [58]

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5 Given the large variability in symptom severity across disease groups, it is difficult to effectively distinguish diseases