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To dissect the upstream regulatory mechanism of miR-29b-3p under Gln deprivation conditions, we integrated transcriptome data (GSE148016) from both Gln+ and Gln- groups, and performed cross-analysis with the predicted upstream transcription factors of miR-29b-3p, ultimately identifying five candidate transcription factors

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Our data are in agreement with a recent report using the same antibody, the specificity of which is suggested by the loss of immunoreactivity in islets from Gcgr / mice [20]