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In addition, ATDC5 cells were induced into cell pellets, as previously reported, and cultured in chondrogenic medium with NPs (50 g mL 1 ) [38]

The supplement CDG is viewed as a potential clinical application for the regulation of estrogen metabolism and as a lipid-lowering agent (it helps maintain normal cholesterol levels)

Notably, deletion of GPX4 caused severe phenotypic abnormalities in mice in animal experiments, whereas deletion of the ALOX12 gene did not cause major developmental defects in mice