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The administration of exendin-4 also regulates S6K activity and the effect is dependent on the activation status of S6K, as occurred with AMPK

In turn, phosphorylation of the adaptor proteins insulin receptor substrate 14 (IRS-14) and Shc leads to activation of the phosphatidyl inositol-3 kinase (PI3K), the mitogen-activated protein kinase (MAPK) and the 14-3-3 pathways (Baserga, 2000)

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The long-term presence of senescent cells has been linked to aging and skin diseases.82 Animal studies have shown that the destruction of senescent cells may prolong survival and reduce carcinogenesis and the incidence of age-related diseases.83 Selective agents targeting senescent cells in the dermis hold promise as a treatment for aging