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Elimination of SLM2 led to a decrease in the non-complementarity of neurexins-SS4- splice variants, with a corresponding loss of strong adhesion and reciprocal binding [68, 80]

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Moreover, EGCG has been shown to suppress the JAK2/STAT3 pathway, thereby reducing the release of pro-inflammatory cytokines such as TNF-, IL-1, and IL-6 in response to inflammatory stimuli like palmitic acid [245]