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Similarly, in the presence of PINK1 mutations, the PINK1 kinase activity will be impaired, which also causes the imbalance of the LRRK2PINK1 kinase pair, leading to disrupted THDA pathway and dopaminergic neuron vulnerability [44]

The number of genes in common with the analysis of any of the HAT inhibitors (C646 + CPTH2, CBP30 + CPTH2, TH1834) was 10 genes ( A3galt2, Acta1, Csrp1, H3f3b, Hebp2, Mki67, Ppdpf, Prtfdc1, Slc16a6, Tuba1c )

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