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Proteomic analysis of mouse cardiac tissue after myocardial infarction induced by left anterior descending ligation demonstrated that the protein levels of the ferroptosis inhibitor GPX4 were significantly downregulated in the early and mid-stages of myocardial infarction

Similarly, 5-methoxytryptophan (5-MTP) inhibits fibroblast-to-myofibroblast transition and ECM protein expression by downregulating TGF-/Smad3 and PI3K/AKT pathways, thereby limiting fibroblast proliferation and migration [113]

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