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The pathogenesis of IBS is associated with disordered gastrointestinal motility, abnormal intestinal secretion, visceral hypersensitivity, altered gut-brain axis, and intestinal permeability, all of which can be affected by the gut microbial community (Pittayanon et al

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This creates an elevated TIMP:MMP ratio that correlates with collagen and elastin synthesis stimulation

This barrier dysfunction allows bacterial products to enter circulation, amplifying systemic inflammation