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Liraglutide exerts anti-catabolic effect in murine primary chondrocytes in vitro To explore the inhibitory effect of liraglutide on the production of catabolic mediators, IL-1-stimulated chondrocytes were cultured with 2 ng/mL of IL-1 and 10 ascending doses (6.6, 13.3, 26.6, 53.1, 106.3, 212.5, 425, 850 nM, 1.7 and 3.4 M) of liraglutide for 24 h for IC 50 determination

Previously, we showed that syndecans control intrinsic cell behavior by regulating transient receptor potential canonical (TRPC) Ca 2+ channels to maintain optimal Ca 2+ influx over the plasma membrane 3

The cyclic approach reflects bioregulator theory that short intensive treatments provide lasting regulatory effects rather than requiring continuous supplementation

Small dense LDL is also more susceptible to glycation even in non-diabetic people [22]