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Together, these findings suggest that SYNCRIP regulates HK2 expression at both the transcriptional and post-transcriptional levels, thereby likely contributing to the modulation of glycolytic activity in GBM cells

Additionally, it was determined that there was a possibility the observed retinal abnormalities were not caused by acute hyperglycemia but rather the loss of IGF-1 activity in cases of diabetes [4]

Genetic variants that alter the function of a therapeutic receptor are known to influence drug response through several mechanisms

The growth inhibitory effects of metformin (Met) and chloroquine (CQ) on the response of NALM-6 cells to cytarabine (AraC) were determined via the MTT assay