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Adding exogenous glutathione to a drug that already modulates oxidative stress raises a conceptual question: could the combined antioxidant load suppress reactive oxygen species (ROS) to a degree that interferes with beneficial ROS signaling
Mitochondrial abnormalities and synaptic damage in huntingtons disease: A focus on defective mitophagy and Mitochondria-Targeted therapeutics
To view a copy of this licence, visit About this article Cite this article Yao, Y., Liu, L., Duan, X
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