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This suggests that some high-molecular-weight peptides might reach enteroendocrine cells and activate GLP-1 secretion, while in other cases the lower-molecular-weight peptides or the amino acids released after digestion are responsible for the secretion

"Glycosylphosphatidylinositol-anchored high-density lipoprotein-binding protein 1 plays a critical role in the lipolytic processing of chylomicrons"

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Combination strategies that integrate antioxidant, anti-inflammatory, epigenetic and neurotrophic approaches, together with approved medications for AUD and structured psychosocial interventions, may offer the greatest potential for preserving brain structure and function [11,21,48]