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In line with this, one other mechanism recently described to be involved in the resistance to GSH depletion is the overexpression of deubiquitinases that inhibit protein degradation following ER-stress (74)

Maintenance of adequate haemodynamic, metabolic, and perfusate homoeostasis enabled successful NMP out to a maximum of 23.4 h

On the other hand, A and tau aggregates can disrupt iron homeostasis, for instance, by interfering with the function of iron-efflux proteins, thereby further intensifying iron accumulation

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