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The antioxidant function of Prdx2 also depends on NADPH

Figure 2 Previous work has demonstrated that aminoglycosides activate acute and slow (chronic) mechanisms that are distinct from one another, with neomycin only activating acute mechanisms but gentamicin activating both pathways (Coffin et al., 2D)

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Preclinical research introduced FOXO4-DRI as a targeted approach for studying senescent cell survival mechanisms by modulating transcription factor interactions rather than global cytotoxic activity